I was allowed to register for ODE without taking the prerequisite (Calc III) though I am taking it at the same time. Will my professor find out? Should I let him know and see if he lets me stay in the class?

Scientifically speaking, how do we solve the frogposting problem?

do you look like a scientist?

Why does noone call scientists out on gatekeeping science?
>hurr durr genetic engineering must be regulated so only my lab can do research

As a society, we often praise individuals for their enormous contributions to science, like how almost everyone has Einstein, Newton, Tesla, Turing, Gauß or Marie Curie in mind when thinking about the stereotypical genius, and I think generally it's a good thing that we elevate those people to such a high degree.
But it also got me thinking, isn't this praising of single individuals kind of a fallacy, because if it weren't for them specifically, eventually (and also likely not longer after the said individual found out), someone else would've found out the same?
Is my premise even right in the first place, or are individual contributions actually as "objectively groundbreaking" and unique as we normally see them?

How really important is math anons? I've been garbage at mate my entire life >currently a NEET
I wanna know how truly important math is.

Assuming we develop a superintelligence, putting the timeframe and feasibility concerns aside, what things would this superintelligence attempt to do and how would it do them?

self preservation, resource acquisition, extermination of humanity, what else? what happens when it achieves these 3 goals?

I tried to improve at Science & Math and got nowhere.
Why does it have to be so hard bros?

>Caffeine is a commonly used neurostimulant that also produces cerebral vasoconstriction by antagonizing adenosine receptors. Chronic caffeine use results in an adaptation of the vascular adenosine receptor system presumably to compensate for the vasoconstrictive effects of caffeine. We investigated the effects of caffeine on cerebral blood flow (CBF) in increasing levels of chronic caffeine use. Low (mean = 45 mg/day), moderate (mean = 405 mg/day), and high (mean = 950 mg/day) caffeine users underwent quantitative perfusion magnetic resonance imaging on four separate occasions: twice in a caffeine abstinent state (abstained state) and twice in a caffeinated state following their normal caffeine use (native state). In each state, there were two drug conditions: participants received either caffeine (250 mg) or placebo. Gray matter CBF was tested with repeated-measures analysis of variance using caffeine use as a between-subjects factor, and correlational analyses were conducted between CBF and caffeine use. Caffeine reduced CBF by an average of 27% across both caffeine states. In the abstained placebo condition, moderate and high users had similarly greater CBF than low users; but in the native placebo condition, the high users had a trend towards less CBF than the low and moderate users. Our results suggest a limited ability of the cerebrovascular adenosine system to compensate for high amounts of daily caffeine use.

>A 250 mg dose of caffeine has been shown to reduce resting cerebral blood flow (CBF) between 22% and 30%

>>Caffeine reduced gray matter CBF by 27% to approximately 60 ml/100 g of tissue/min across all caffeine users

How can someone justify using caffeine after seeing stuff like this? It's a vasoconstrictor, it'll turn your brain into mush.

>has no comfortable sleeping position without tools