Brew tea for 2 mins: caffeine it released
Brew tea for 5 mins: theanine and tannins are released which neutralize the caffeine
Drink 5+ min brewed tea on an empty stomach, you'll get nausea and perhaps vomit, due to the excess tannins being taken in too quickly.
>Theanine is structurally similar to the excitatory neurotransmitter glutamate, and in accordance, binds to glutamate receptors, though with much lower affinity in comparison. Specifically, it binds to ionotropic glutamate receptors in the micromolar range, including the AMPA and kainate receptors and, to a lesser extent, the NMDA receptor.[15][16][17][18] It acts as an antagonist of the former two sites[18] and as an agonist of the latter site.[19] Theanine also binds to group I mGluRs.[15][20] In addition, it inhibits glutamine transporters and glutamate transporters, and thus blocks the reuptake of glutamine and glutamate.[17][21][22]
https://en.wikipedia.org/wiki/Theanine#Pharmacology
>Excitotoxicity due to excessive glutamate release and impaired uptake occurs as part of the ischemic cascade and is associated with stroke,[9] autism,[10] some forms of intellectual disability, and diseases such as amyotrophic lateral sclerosis, lathyrism, and Alzheimer's disease.[9][11] In contrast, decreased glutamate release is observed under conditions of classical phenylketonuria[12] leading to developmental disruption of glutamate receptor expression.[13]
https://en.wikipedia.org/wiki/Glutamate_(neurotransmitter)#Disease,_disabilities,_and_pharmacology
I wish there was less bullshit to wade through when doing searches on this stuff. I'm sick to shit of blogs, books, and bullshit in google scholar searches.
Brew tea for 5 mins: theanine and tannins are released which neutralize the caffeine
Drink 5+ min brewed tea on an empty stomach, you'll get nausea and perhaps vomit, due to the excess tannins being taken in too quickly.
>Theanine is structurally similar to the excitatory neurotransmitter glutamate, and in accordance, binds to glutamate receptors, though with much lower affinity in comparison. Specifically, it binds to ionotropic glutamate receptors in the micromolar range, including the AMPA and kainate receptors and, to a lesser extent, the NMDA receptor.[15][16][17][18] It acts as an antagonist of the former two sites[18] and as an agonist of the latter site.[19] Theanine also binds to group I mGluRs.[15][20] In addition, it inhibits glutamine transporters and glutamate transporters, and thus blocks the reuptake of glutamine and glutamate.[17][21][22]
https://en.wikipedia.org/wiki/Theanine#Pharmacology
>Excitotoxicity due to excessive glutamate release and impaired uptake occurs as part of the ischemic cascade and is associated with stroke,[9] autism,[10] some forms of intellectual disability, and diseases such as amyotrophic lateral sclerosis, lathyrism, and Alzheimer's disease.[9][11] In contrast, decreased glutamate release is observed under conditions of classical phenylketonuria[12] leading to developmental disruption of glutamate receptor expression.[13]
https://en.wikipedia.org/wiki/Glutamate_(neurotransmitter)#Disease,_disabilities,_and_pharmacology
I wish there was less bullshit to wade through when doing searches on this stuff. I'm sick to shit of blogs, books, and bullshit in google scholar searches.
