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Now the main question is:
Imagine you get the mRNA vaccine and its sequence (I didn't mention it in the previous paragraph but the binding to your endogenous mRNA is obviously sequence-specific, otherwise it could just randomly bind to anything and not lead to the expected result) would match the sequence of some of your endogenous mRNAs and with some (although extremely low) probability it could bind to one of the sites I mentioned (ribosome binding site or splicing regions) and cause lower protein expression
Technically it wouldn't really do much, because if it's not really an important gene, you would get a lower expression for a certain amount of time, but in the end, the mRNA from the vaccine would get degraded and you might not notice a difference and the mRNA from the vaccine would be translated into protein which would induce an immune response, you create a bunch of memory T-cells and all is good.
But here's the bad scenario, mRNA from the vaccine binds to endogenous mRNA in your cells that encodes some essential gene, specifically some tumor suppressor gene. It has been confirmed that loss of tumor suppressor protein expression leads to the transformation of your cells into cancer cells. Even though the vaccine mRNA would stay in your system for a very small amount of time, this can be sufficient for some cells to become cancer cells and after that, it's purely a waiting game because at this stage you can't really locate cancer cells, and of course, the development of cancer is a lengthy process so it can take ten years before we figure out if anything happened, so for older people who took the vaccine, it would probably be never taken into account.
