/sci/ - Science & Math » Thread #11527812

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Anonymous Fri 03 Apr 02:38:58 2020 No.11527812 View Reply Original Report

Quoted By: >>11527859 >>11527890 >>11527942 >>11528590 >>11530805 >>11530831 >>11530850

Can any medfags debunk this?

Anonymous

Anonymous Fri 03 Apr 2020 02:48:44 No.11527837 Report

Quoted By: >>11530805

it's all bullshit

Anonymous

Anonymous Fri 03 Apr 2020 02:56:02 No.11527848 Report

Quoted By:

>Anti-HIV drugs works
[citation needed]
We so far have no anti-virals against SARS-CoV-2 whatsoever.

Anonymous

Anonymous Fri 03 Apr 2020 03:03:54 No.11527859 Report

Quoted By: >>11527866

>>11527812
why didn't you just post this in your other thread
>>11526932

Anonymous

Anonymous Fri 03 Apr 2020 03:10:14 No.11527866 Report

Quoted By:

>>11527859
I will it's my first time posting here

Anonymous

Anonymous Fri 03 Apr 2020 03:21:27 No.11527890 Report

Quoted By:

>>11527812
He states that ACE2 receptors determine the speed of colonization, which is why Asia seemed to get hit first (higher expression of ACE2 receptors). He also states that children are going to be affected last because it's taking longer to suppress their immune systems. However, ACE2 expression is highest in children, and declines with age. Also, children handled SARS better than adults as well, and it worked through a similar mechanism.

https://www.the-scientist.com/news-opinion/possible-biological-explanations-for-kids-escape-from-covid-19-67273

>SARS-CoV and SARS-CoV-2 both use the same keyhole to enter cells, the ACE2 receptor. There’s an abundance of this receptor in cells in the lower lung, which may explain the high incidence of pneumonia and bronchitis in those with severe COVID-19 infection. A recent study showed that ACE2 is also highly expressed in the mouth and tongue, granting the virus easy access to a new host. ACE2 receptor abundance goes down in the elderly in all these tissues, but, counterintuitively, this might place them at a greater risk of severe illness.

>This is because the ACE2 enzyme is an important regulator of the immune response, especially inflammation. It protects mice against acute lung injury triggered by sepsis. And a 2014 study found that the ACE2 enzyme offers protection against lethal avian influenza. Some patients with better outcomes had higher levels of the protein in their sera, and turning off the gene for ACE2 led to severe lung damage in mice infected with H5N1, while treating mice with human ACE2 dampened lung injury.

>A fall in ACE2 activity in the elderly is partly to blame for humans’ poorer ability to put the brakes on our inflammatory response as we age, according to emailed comments from Hongpeng Jia of Johns Hopkins Medicine. Reduced abundance of ACE2 receptors in older adults could leave them less able to cope with SARS-CoV-2, says Baric, though the hypothesis still needs more research.

Anonymous

Anonymous Fri 03 Apr 2020 03:43:17 No.11527942 Report

Quoted By:

>>11527812
Person in OPs picture has no idea what he is talking about, and does not understand biology chemistry or physics

Anonymous

Anonymous Fri 03 Apr 2020 04:04:06 No.11527990 Report

Quoted By:

They dont even know what antibodies are, the larp falls apart in the first paragraph

Anonymous

Anonymous Fri 03 Apr 2020 09:40:34 No.11528590 Report

Quoted By:

>>11527812
>The virus will infect your antibodies

Anonymous

Anonymous Fri 03 Apr 2020 09:51:38 No.11528601 Report

Quoted By:

>Corona was released
Yeah, you can safely stop reading there
>Antibodies become infected
I warned you.

It's drivel.

Anonymous

Anonymous Fri 03 Apr 2020 14:06:00 No.11529224 Report

Quoted By: >>11529338

Where the fuck did this "COVID-19 is secretly AIDS" nonsense spawn from?

I can't tel if it was retarded doomer bloggers and /pol/ picked up on it or if it was retards from /pol/ and then doomer bloggers took it from there.

Anonymous

Anonymous Fri 03 Apr 2020 14:53:20 No.11529338 Report

Quoted By: >>11529355 >>11529362

>>11529224
>Where the fuck did this "COVID-19 is secretly AIDS" nonsense spawn from?
https://www.researchgate.net/publication/338957445_Uncanny_similarity_of_unique_inserts_in_the_2019-nCoV_spike_protein_to_HIV-1_gp120_and_Gag

"We are currently witnessing a major epidemic caused by the 2019 novel coronavirus (2019- nCoV). The evolution of 2019-nCoV remains elusive. We found 4 insertions in the spike glycoprotein (S) which are unique to the 2019-nCoV and are not present in other coronaviruses. Importantly, amino acid residues in all the 4 inserts have identity or similarity to those in the HIV-1 gp120 or HIV-1 Gag. Interestingly, despite the inserts being discontinuous on the primary amino acid sequence, 3D-modelling of the 2019-nCoV suggests that they converge to constitute the receptor binding site. The finding of 4 unique inserts in the 2019-nCoV, all of which have identity /similarity to amino acid residues in key structural proteins of HIV-1 is unlikely to be fortuitous in nature. This work provides yet unknown insights on 2019-nCoV and sheds light on the evolution and pathogenicity of this virus with important implications for diagnosis of this virus."

Anonymous

Anonymous Fri 03 Apr 2020 15:00:14 No.11529355 Report

Quoted By: >>11529563

>>11529338
Also, here's where the paper was originally posted: https://doi.org/10.1101/2020.01.30.927871

"This paper has been withdrawn by its authors. They intend to revise it in response to comments received from the research community on their technical approach and their interpretation of the results. If you have any questions, please contact the corresponding author."

And here's a paper that refutes the findings: https://doi.org/10.1021/acs.jproteome.0c00129

Anonymous

Anonymous Fri 03 Apr 2020 15:03:03 No.11529362 Report

Quoted By:

>>11529338
>only research paper that claimed this
>pulled and refuted by several others
>complete misunderstanding on the part of moronic laymen /pol/shit doomers looking for anything to justify their insane theories of what in HIV allows it to be a chronic illness
So it's just retardation then? About what I figured.

Anonymous

Anonymous Fri 03 Apr 2020 16:18:50 No.11529563 Report

Quoted By: >>11529871

>>11529355
At a first glance, it refutes their claim that these insertions were unique to HIV-1. Not that they exist.
So this increases the likelihood of it having happened by mutation, but doesn't mean it wasn't one of the strains where they deliberately put together parts of HIV, SARS, and bat Coronavirus. I think the odds are still higher that the BSL-4 lab was managed shittily and it came from there.

Anonymous

Anonymous Fri 03 Apr 2020 18:04:19 No.11529871 Report

Quoted By: >>11530439

>>11529563
The original paper was essentially arguing that it had to be a man-made virus (unlikely to be fortuitous in nature"), which is not the case, and of course comparing it to HIV garnered plenty of attention. It was scientists being irresponsible in an effort to make a name for themselves, and then the public misconstrued the information.

Anonymous

Anonymous Fri 03 Apr 2020 21:34:14 No.11530439 Report

Quoted By: >>11530506

>>11529871
Explain the fact that it has 4 spike proteins from HIV

There’s no way that happened in nature

Anonymous

Anonymous Fri 03 Apr 2020 22:18:42 No.11530506 Report

Quoted By: >>11530611 >>11530758

>>11530439
>Explain the fact that it has 4 spike proteins from HIV
It doesn't, it has several spike proteins that are similar. Whether you want to call it 1 or 4 depends on what degree you want to grade the similarity. Other viruses develop similar proteins as well. They identified another coronavirus, RaTG13, which is far more similar to SARS-CoV-2 in terms of the four spike proteins the first study identified.

Anonymous

Anonymous Fri 03 Apr 2020 23:11:02 No.11530611 Report

Quoted By:

>>11530506
SHUT THE FUCK UP, SHILL
REEEEEEEEEEEEEEEEEEEEEEEEEE

Anonymous

Anonymous Sat 04 Apr 2020 00:10:52 No.11530758 Report

Quoted By:

>>11530506
It does

Anonymous

Anonymous Sat 04 Apr 2020 00:24:37 No.11530805 Report

Quoted By:

>>11527812
>was released
No it wasn’t.
>COVID-19 is an immun suppressant
No, first the virus is called SARS-Cov-2 or formerly nCov-2019. COVID-19 is the illness it causes. All viruses have developed some amount of tricks to prevent the immune system from immediately „destroying“ it, but SARS-Cov-2 isn’t that good. In fact the immun response due to the medium level tropism is quiet substantial and there is no sign of it significantly having anything to offer against it. In fact in some cases the innate immun system seems to defeat the infection alone, since some have the PCR positiv result, but signs, symptoms and sharing of virions stop, without an anti-body response. >>11527837

Anonymous

Anonymous Sat 04 Apr 2020 00:35:03 No.11530831 Report

Quoted By:

>>11527812
>it binds to ACE2
Partly true it has a affinity to that receptor. There could be other receptors that could play a role at enhancing it or make entry possible themselves.
>R0 equals 9
Coronaviridae aren’t build that way, staying in the air meaning spreading on particles like measles isn’t possible for them, they have 29.000 base pairs and a „fat“ hull, they will drop rather soon. Anything above R0 equaling 6 is almost impossible. Even that could only be reached, if there is an enormous amount of smear infection too. Which isn’t really shown in humanCoronaviruses.
Can somebody write the picture out, because typing everything in and answering will take days.

Anonymous

Anonymous Sat 04 Apr 2020 00:43:38 No.11530850 Report

Quoted By:

>>11527812
>you are already infected
We are currently figuring this out with antibody testing.
>it takes time to destroy a kids immun system
In fact that’s just wrong influenza is probably the virus that pulls the most tricks on the human immune system and we can clearly see it hits children harder on average. Especially young once, it’s slightly more complicated with pandemic influenza, but small children are still hit very hard.
>critical level of virus after months
Looking at the Munich cohort this is clearly nonsense they were tested multiple times after being in contact to infected individuals. And if they got it their nasal swab was positive in PCR testing after no more than a couple days.

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