This is a guess, but it really depends on what model they're assuming. You can have hepatic lipotoxicity without any adipocytic involvement (e.g., a choline deficiency), elevated EGP from elevating circulating FFAs alone without any inflammation in-between (
https://www.cell.com/cell/pdfExtended/S0092-8674(15)00014-8), hyperglycemia without any elevated EGP (if skeletal muscle alone becomes sufficiently insulin resistant), NAFLD with sterols instead of fatty acids (
https://www.sciencedirect.com/science/article/abs/pii/S1043276015002337)
Here's a good review article that you might want to read
https://www.cell.com/action/showPdf?pii=S0092-8674%2813%2901546-8 
