219KiB, 1000x1495, 4a0a70f2b0d545be9f73fa405465a0d8730fa3f51da87d9b052147cf18f86f2d.jpg
You Wouldn't Patent the?Sun
No.14148915 ViewReplyOriginalReport
Quoted By: >>14148929 >>14150231 >>14150335
Vindicated by Gwangji Institute of Science and Technology research...
https://8kun dot top/x/res/60849.html
https://www.sciencedaily.com/releases/2022/01/220124103914.htm
This recent study by Gwangju Institute of Science and Technology provides reinforcement for my theory of the behavior of both Alzheimer's Disease and the nature of memory formation and retrieval.
Please see:
https://8kun dot top/x/res/60049.html
Taken together, 21Jul2021 (60049) and this thread's (15Jan2022, 60849) concepts are vindicated by this new report.
I speculate that the Gwangju experiment was successful in slowing Alzheimer's progression because the ultrasound waves changed the operating frequency of the brain and because memories are encoded in different regions and on different protein strands depending upon frequency. Since Alzheimers progresses due to the increasing length and volume of the amyloid plaques (amyloids being the proteins in which memories are encoded) this change in frequency prevents any new memories from being added to the malfunctioning areas with plaques.
It makes sense that anything that causes the brain to create memories in non-standard strands would help it to avoid the further lengthening of the plaques. This is not a cure (nor is my proposition for boosting levels of concatenator proteins after TBIs,) however, this would seem to suggest that my model of memory formation is the correct one and only further demonstrates the potential value of investigating my theories.
https://8kun dot top/x/res/60849.html
https://www.sciencedaily.com/releases/2022/01/220124103914.htm
This recent study by Gwangju Institute of Science and Technology provides reinforcement for my theory of the behavior of both Alzheimer's Disease and the nature of memory formation and retrieval.
Please see:
https://8kun dot top/x/res/60049.html
Taken together, 21Jul2021 (60049) and this thread's (15Jan2022, 60849) concepts are vindicated by this new report.
I speculate that the Gwangju experiment was successful in slowing Alzheimer's progression because the ultrasound waves changed the operating frequency of the brain and because memories are encoded in different regions and on different protein strands depending upon frequency. Since Alzheimers progresses due to the increasing length and volume of the amyloid plaques (amyloids being the proteins in which memories are encoded) this change in frequency prevents any new memories from being added to the malfunctioning areas with plaques.
It makes sense that anything that causes the brain to create memories in non-standard strands would help it to avoid the further lengthening of the plaques. This is not a cure (nor is my proposition for boosting levels of concatenator proteins after TBIs,) however, this would seem to suggest that my model of memory formation is the correct one and only further demonstrates the potential value of investigating my theories.
