do you guys think this might actually be real
The spike protein sans virus (i.e. as produced or delivered by all vaccines, mRNA, viral vector, or otherwise), once bound to ACE2 receptors, causes the body to activate viral genetic code present in our DNA, resulting in production of a protein called HERV-W.
>the presently observed HERV activation is not induced by cytokines, or by inflammation due to viral infection, but rather by SARS-CoV-2 spike protein itself.
>[This study] revealed that SARS-CoV-2 via its spike protein may trigger HERV expression in human PBMC[...]
>Charvet, B. et al, 2021, SARS-CoV-2 induces transcription of human endogenous retrovirus RNA followed by type W envelope protein expression in human lymphoid cells.
This HERV-W is then identified by white blood cells, and in turn, they begin to produce auto-antibodies against HERV-W.
>[Here] we have analyzed HERV-H Env and HERV-W Env epitope expression on the surface of PBMCs [and have] analyzed serum antibody levels to the expressed HERV-H and HERV-W Env epitopes [the PBMCs had] higher antibody reactivities towards HERV-H Env and HERV-W Env epitopes.
>The higher antibody reactivities [...] correlate with the higher levels of HERV-H Env and HERV-W Env expression on B cells and monocytes.
>Brudek, T., Christensen, T., Aagaard, L., Petersen, T., Hansen, H. and Møller-Larsen, A., 2009. B cells and monocytes from patients with active multiple sclerosis exhibit increased surface expression of both HERV-H Env and HERV-W Env, accompanied by increased seroreactivity. Retrovirology, 6(1).
https://archive.4plebs.org/pol/thread/351764743/#q351768036
The spike protein sans virus (i.e. as produced or delivered by all vaccines, mRNA, viral vector, or otherwise), once bound to ACE2 receptors, causes the body to activate viral genetic code present in our DNA, resulting in production of a protein called HERV-W.
>the presently observed HERV activation is not induced by cytokines, or by inflammation due to viral infection, but rather by SARS-CoV-2 spike protein itself.
>[This study] revealed that SARS-CoV-2 via its spike protein may trigger HERV expression in human PBMC[...]
>Charvet, B. et al, 2021, SARS-CoV-2 induces transcription of human endogenous retrovirus RNA followed by type W envelope protein expression in human lymphoid cells.
This HERV-W is then identified by white blood cells, and in turn, they begin to produce auto-antibodies against HERV-W.
>[Here] we have analyzed HERV-H Env and HERV-W Env epitope expression on the surface of PBMCs [and have] analyzed serum antibody levels to the expressed HERV-H and HERV-W Env epitopes [the PBMCs had] higher antibody reactivities towards HERV-H Env and HERV-W Env epitopes.
>The higher antibody reactivities [...] correlate with the higher levels of HERV-H Env and HERV-W Env expression on B cells and monocytes.
>Brudek, T., Christensen, T., Aagaard, L., Petersen, T., Hansen, H. and Møller-Larsen, A., 2009. B cells and monocytes from patients with active multiple sclerosis exhibit increased surface expression of both HERV-H Env and HERV-W Env, accompanied by increased seroreactivity. Retrovirology, 6(1).
https://archive.4plebs.org/pol/thread/351764743/#q351768036
